Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. However, our ability to rescue SERT purpose in KI midbrain synaptoneurosomes with the inhibition of FAK signifies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by increased pFAK localization in five-HT synapses. Our info https://pro33-login66655.ltfblog.com/31347090/fascination-about-pro33