Preliminary studies indicate conolidine may possibly inhibit unique ion channels, cutting down neuronal excitability and restricting agony signals. This mechanism is particularly relevant in neuropathic soreness, wherever abnormal signaling will cause persistent discomfort. In addition, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to discomfort perc... https://johnk294agx6.blogars.com/profile